Multiple Sclerosis, Mad Cow Disease and Acinetobacter by Alan Ebringer

By Alan Ebringer

The target of this booklet is to publicise and produce to a much broader viewers the idea that the reason for neurological illnesses, particularly a number of sclerosis (MS) and “mad cow affliction” often referred to as “bovine spongiform encephalopathy” are similar via publicity to a typical microbe Acinetobacter that is present in human sinuses, on pores and skin and within the soil. infection is the reason for a neurological affliction in guy and in animals. increased degrees of antibodies to Acinetobacter were present in a number of sclerosis sufferers in addition to in ruminants who've been defined as being affected by “mad cow ailment” following publicity to infected feed vitamins. the final target and scope of this e-book is to notify the viewers, the reader, that a number of sclerosis should be associated with a microbe Acinetobacter which consists of molecular constructions corresponding to myelin, the outer sheath overlaying of neurons.

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Extra info for Multiple Sclerosis, Mad Cow Disease and Acinetobacter

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4 5 BSE: Comparison Between the “Prion” Hypothesis and the Autoimmune Theory Difficulties Associated with the Prion Hypothesis There are several problems associated with the concept that prions are the causative agents of BSE: 1. The prion hypothesis has not been able to account for the different types of PrPsc which have been detected in scrapie brains. Since PrPs do not contain any nucleic acids, genetic mutations cannot explain the occurrence of these different forms of prions which have different incubation times and also various types of rod shapes.

The rabies virus travels to the brain by following the peripheral nerves. The incubation period of the disease is usually a few months in humans, depending on the distance the virus must travel, from the site of the bite to reach the central nervous system. Once the rabies virus reaches the central nervous system and symptoms begin to show, the infection is virtually untreatable and usually fatal within days to weeks. Early-stage symptoms of rabies are malaise, headache and fever, progressing to acute, violent movements, uncontrolled excitement, depression and a characteristic hydrophobia due to pharyngeal spasm brought on by attempts to drink.

The question still remains as to why this ban has been successful. Was it due to the elimination of animals infected by prions or could another explanation account for these results.

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