By Roger Hull
It's been ten years because the booklet of the 3rd variation of this seminal textual content on plant virology, within which there was an explosion of conceptual and authentic advances. The fourth version updates and revises many information of the former editon, whereas conserving the $64000 older effects that represent the field's conceptual starting place. Key beneficial properties of the fourth version include:* Thumbnail sketches of every genera and relations teams* Genome maps of all genera for which they're recognized* Genetic engineered resistance concepts for virus sickness keep an eye on* most recent figuring out of virus interactions with crops, together with gene silencing* Interactions among viruses and bug, fungal, and nematode vectors* New plate part containing over 50 full-color illustrations
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Additional info for Matthews' Plant Virology, Fourth Edition
Different viruses m a y cause very similar s y m p t o m s on the same host plant. 3. Some diseases m a y be caused by a mixture of two unrelated viruses. I. N O M E N C L A T U R E A. Historical aspects In all studies of natural objects, h u m a n s have an innate desire to n a m e and to classify. Virologists are no exception. Virus classification, as with all other classifications, arranges objects showing similar properties into groups and, even t h o u g h this m a y be a totally artificial and h u m a n driven activity without any natural base, it does have certain properties: J.
2). There are 14 families recognized for plant viruses (Appendix 2); some, such as Reoviridae and Rhabdoviridae, are in c o m m o n with animal virus families. Twenty two of the genera have not yet been assigned to families and are termed 'floating genera'. The acquisition of further data on these 'floating genera', together with changing attitudes on virus classification, will no doubt lead to the designation of further plant virus families. g. g. Geminiviridae, descriptive of the virus particles).
3. G e n o t y p i c i n f o r m a t i o n is n o w m o r e i m p o r tant for m a n y aspects of virus t a x o n o m y t h a n p h e n o t y p i c characters. H o w e v e r , there are s o m e limitations to the use of g e n o t y p e (sequence) d a t a alone. A n i m p o r t a n t one is that w i t h the p r e s e n t state of k n o w l e d g e it is v e r y difficult, or impossible, to predict p h e n o t y p i c p r o p e r t i e s of a virus b a s e d on sequence data alone. For e x a m p l e , if w e h a v e t w o viral n u c l e o t i d e sequences differing in a nucleotide at a single site, w e could not, in m o s t cases, d e d u c e f r o m this i n f o r m a t i o n alone that one led to m o s a i c disease a n d the other to lethal necrosis in the s a m e host.