Ciclosporin in Autoimmune Diseases: 1st International by R. Schindler (auth.), Mrs. Rosemarie Schindler (eds.)

By R. Schindler (auth.), Mrs. Rosemarie Schindler (eds.)

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Additional info for Ciclosporin in Autoimmune Diseases: 1st International Symposium, Basle, March 18–20, 1985

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However, these homo- old observations that most autoimmune zygous deficiencies of a complement com- diseases are heavily biased towards females ponent account for only a very small pro- indicates another genetically based cause of portion of idiopathic SLE encountered in susceptibility which deserYes further investigation. practice. The second piece of evidence is that idioLirnitations of space have also prevented pathic SLE has been reported to be as- a proper discussion of autoimmune resociated with several HLA antigens [47].

Penicillamine-indueed It is tempting to speculate that the Class myasthenia gravis in rheumatoid III genes are particularly likely to be inarthritis patients volved in genetic susceptibility to diseases 22 renal damage arising in patients with rheumatoid arthritis, the association with the haplotype carrying HLA-B8, CW7, and DR3 is very strong; approximately 50% of patients with gold nephropathy have this haplotype, and more than 90% with this haplotype develop nephropathy if given a full course of gold therapy.

Lilly F, Boyse EA, Old U (1964) Lancet ii: 1207 24. McDevitt HO, Sela M (1965) J Exp Med 122:517 25. McDevitt HO, Chinitz A (1969) Science 163: 1207 26. McMichael AJ (1978) J Exp Med 148: 1458 27. Nishimura Y, Sasazuki T (1983) Nature 302:67 28. Parham P (1984) Immunol Today 5:89 29. Perry RM (1973) Am J Med 54:58 30. Pinchuck P, Maurer PH (1965) J Exp Med 122:673 31. Ploegh HL, Orr RT, Strominger JL (1981) Ce1l24:287 32. Pober JS, Gimbrone MA, Cotran RS, Reiss CS, Burakoff SJ, Fiers W, Ault KA (1983) J Exp Med 157: 1339 33.

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